Synonyymit & Tietoja | englanti sana THYROTROPIN

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Esimerkkejä THYROTROPIN käyttämisestä lauseessa

• Thyroid-stimulating hormone (TSH or thyrotropin) – stimulates the thyroid gland to make and release thyroid hormone.
• Results revealed that in the sleep-debt condition, thyrotropin concentrations were decreased, while lowered glucose and insulin responses indicated a clear impairment of carbohydrate tolerance, a 30% decrease than in the well-rested sleep condition.
• Cyclic breast pain is often associated with fibrocystic breast changes or duct ectasia and thought to be caused by changes of prolactin response to thyrotropin.
• A TFT panel typically includes thyroid hormones such as thyroid-stimulating hormone (TSH, thyrotropin) and thyroxine (T4), and triiodothyronine (T3) depending on local laboratory policy.
• Ions such as thiocyanate (from cigarette smoking for example) and perchlorate decrease iodide uptake by competitive inhibition and, as a consequence of reduced thyroxine and triiodothyronine secretion by the gland, cause, at low doses, an increased release of thyrotropin (by reduced negative feedback), which then stimulates the gland.
• The thyrotropin receptor (or TSH receptor) is a receptor (and associated protein) that responds to thyroid-stimulating hormone (also known as "thyrotropin") and stimulates the production of thyroxine (T4) and triiodothyronine (T3).
• The pituitary gland secretes thyrotropin (TSH; Thyroid Stimulating Hormone) that stimulates the thyroid to secrete thyroxine (T4) and, to a lesser degree, triiodothyronine (T3).
• Neuroendocrinology of Prolonged Critical Illness: Effects of Exogenous Thyrotropin Releasing Hormone and Its Combination with Growth Hormone Secretagogues.
• The most clinically relevant anti-thyroid autoantibodies are anti-thyroid peroxidase antibodies (anti-TPO antibodies, TPOAb), thyrotropin receptor antibodies (TRAb) and thyroglobulin antibodies (TgAb).
• SimThyr was also used to show that the release rate of thyrotropin is controlled by multiple factors other than T4 and that the relation between free T4 and TSH may be different in euthyroidism, hypothyroidism and thyrotoxicosis.
• After receiving her doctorate in 1965 at the University of Munich with a dissertation titled "Veränderungen des Sekretintestes bei subaciden, cholecystektomierten und pankreaskranken Patienten" (Changes of the secretin test in subacid patients after cholecystectomy and with pancreatic diseases) she habilitated in 1972 at the Ludwig-Maximilian University with a treatise titled "Stimulation der TSH-Sekretion durch TRH (Thyrotropin Releasing Hormone): diagnostische Bedeutung und pathophysiologische Folgerungen" (Stimulation of thyrotropin secretion by TRH (thyrotropin releasing hormone): diagnostic utility and pathophysiological consequences).

Sivun valmistelu kesti: 230,47 ms.