Informatie over | Engels woord ISCHAEMIA

Voorbeelden van het gebruik van ISCHAEMIA in een zin

• If they are shunted to the systemic circulation through a patent foramen ovale they can travel to and lodge in the brain where they can cause stroke, the coronary capillaries where they can cause myocardial ischaemia or other tissues, where the consequences are usually less critical.
• She died on 31 January 2004, after suffering from various ailments, including hypoglycemia, ischaemia and insulinoma.
• Present-day analysis shows that they can be composed of coagulated fibrin including platelets, focal ischaemia, inflammatory infiltrate, infectious organisms, or neoplastic cells.
• Rates of postoperative spinal cord ischaemia can be kept at low levels after open surgical repair with the adequate precautions and perioperative maneuvers.
• Due to splanchnic hypoperfusion and the subsequent mucosal ischaemia there are structural changes and alterations in cellular function.
• Vegetations occurring in the context of Libman–Sacks endocarditis may dislodge to form emboli and cause embolism (including cerebral embolism (presenting as stroke or transient ischaemic attack), mesenteric ischaemia (presenting with severe abdominal pain), or peripheral arterial embolism (presenting with limb coldness)).
• It is useful in the diagnosis and treatment of arterial and venous occlusions, including carotid artery stenosis, pulmonary embolisms, and acute limb ischaemia; arterial stenosis, which is particularly useful for potential kidney donors in detecting renal artery stenosis (DSA is the gold standard investigation for renal artery stenosis); cerebral aneurysms and arteriovenous malformations (AVM).
• The CM5 Electrocardiography lead configuration (right arm electrode on manubrium, left arm electrode on V5 and indifferent lead on left shoulder), used to detect left ventricular ischaemia during general anaesthesia.
• For neurologic features, those who have FES may become lethargic, restless, with a drop in Glasgow Coma Scale (GCS) due to cerebral oedema rather than cerebral ischaemia.
• In the heart, IPC is an intrinsic process whereby repeated short episodes of ischaemia protect the myocardium against a subsequent ischaemic insult.
• The drug inhibits the vicious circle of myotonia by decreasing pain, ischaemia, and hypertonia in skeletal muscles, thus alleviating stiffness and spasticity, and facilitating muscle movement.
• Infections, chronic or acute, can affect blood flow by inducing platelet activation and aggregation, contributing to a localized state of excess coagulability (hypercoagulability) that may contribute to clot formation (thrombosis), a known cause of bone infarct and ischaemia.
• The interaction site of emopamil suggests to its greater neuroprotective efficacy in research related to ischaemia.
• Blue toe syndrome is a situation that may reflect atherothrombotic (clots resulting from the build-up of fats, cholesterol, and other substances in and on the artery walls) microembolism, causing transient focal ischaemia, a temporary blockage of blood flow to the brain or spinal cord, occasionally with minor apparent tissue loss, but without diffuse forefoot ischemia.
• Efonidipine increases coronary blood flow by blocking L & T-type calcium channels and attenuates myocardial ischaemia.
• CMR perfusion is increasingly used in cardiac imaging to test for inducible myocardial ischaemia and has been well validated against other imaging modalities such as invasive angiography or FFR.
• The increased risk of mortality associated with high concentrations and the lack of efficacy at low concentration make it unlikely that cariporide will reach market as a cardioprotective agent during ischaemia and reperfusion.
• A detailed analysis of the phenomenon of delayed anti-anginal action revealed that PgI2-pretreatment protected against consequences of ischaemia, such as early morphological changes, early and late postocclusion and reperfusion arrhythmias due to coronary artery occlusion or ouabain intoxication.
• the initial 4-6 hour period of ischemic preconditioning, known as classical or early preconditioning, in which a coronary occlusion can limit injuries of a subsequent ischaemia reperfusion in the myocardium.
• the period of ischemic preconditioning, starting 24 hours after a coronary occlusion and lasting up to 72 hours, that can limit injuries of a subsequent ischaemia reperfusion in the myocardium.
• Thrombosis within the aneurysm and subsequent luminal narrowing may result in claudication of gradual onset, while an acute thrombosis (occluding the vessel at the side of the aneurysm or lodging distally as the vessel narrows) may lead to acute lower extremity ischaemia and associated symptomatology (pain, paresthesia, paresis, pallor, poikilothermia).
• "Characterization of the excitation—contraction coupling system of the hypothermic myocardium following ischaemia and repercussion".
• After subarachnoidal bleedings, irritation of the blood vessels can lead to a vasospasm and thus to an ischaemia, an insufficient blood supply to brain tissue.
• Diseases in aorta are recognized as a widely seen contributor of spontaneous spinal cord ischaemia, represented by rupturing of thoracic aortic aneurysm, arterial occlusion by aortic intima separated from endothelial wall in aortic dissection, and aortic coarctation.

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